Frequency of epicardial coronary lesions in patients with Dilated Heart Disease in a Department of Cardiology in the period from 2015 to 2021
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Abstract
Introduction: dilated heart disease defined as the dilation of cardiac cavities of multiple etiologies has a prevalence in adults of around 1/2.500 individuals. Transthoracic echocardiography determines chamber dilation and evaluates ventricular function, while coronary angiography can identify the mainly ischemic etiology due to involvement of the epicardial coronary arteries. The use of coronary angiography as the first line for the etiological diagnosis implies risks of an invasive method with a high cost in economic resources, for which it is imperative to determine the possible result of said procedure.
Objective: to determine the frequency of epicardial coronary lesions in patients with Dilated Heart Disease in a Departmento de Cardiología in the period 2015-2021.
Methodology: observational, descriptive, retrospective and cross-sectional study with non-probabilistic convenience sampling, by reviewing the database in a Departmento de Cardiología.
Results: this study included 88 patients, with dyspnea in NYHA functional class II-IV and dilated heart disease by echocardiography, of unknown etiology. Out of this group, 66% were men and 34 % women, the mean age was 56 ± 11.19 years. When undergoing diagnostic cinecoronariography, it was found that 23 % had epicardial coronary lesions, the most frequent being a lesion of a vessel (55 %), with the male sex predominating in those with or without epicardial coronary lesions.
Conclusions: the frequency of epicardial coronary lesions in patients with dilated heart disease islow.
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References
Bart BA, Shaw LK, McCants CB, Fortin DF, Lee KL, Califf RM, et al. Clinical determinants of mortality in patients with angiographically diagnosed ischemic or nonischemic cardiomyopathy. J Am Coll Cardiol. 1997;30(4):1002–8. doi: 10.1016/s0735-1097(97)00235-0.
Shore S, Grau-sepulveda MV, Bhatt DL, Heidenreich PA, Eapen ZJ, Hernandez AF, et al. Characteristics, Treatments, and Outcomes of Hospitalized Heart Failure Patients Stratified by Etiologies of Cardiomyopathy. JACC Heart Fail. 2021(3)11;1–11.
Braunwald E. Cardiomyopathies: an overview. Circ Res. 2017;121(7):711–21. doi: 10.1161/CIRCRESAHA.117.311812.
Hänselmann A, Veltmann C, Bauersachs J, Berliner D. Dilated card omyopathies and non-compaction cardiomyopathy. Herz. 2020;45(3):212-220. doi: 10.1007/s00059-020-04903-5.
Alves AB, Danilo, Bastos P, Aparecida D, Silva D. Beaumont, et. al. 2007. Acta Biomedica Brasiliencia [Internet]. 2018;5(2):387–98. Dispobible en: www.actabiomedica.com.br
Nadruz W. Myocardial remodeling in hypertension. Journal of Human Hipertension. 2014;29(1):1–6. doi:10.1038/jhh.2014.36
Slivnick J, Lampert BC. Hypertension and heart failure. Heart Fail Clin [Internet]. 2019;15(4):531–41. doi:10.1016/j.hfc.2019.06.007
Bozkurt B, Colvin M, Cook J, Cooper LT, Deswal A, Fonarow GC, et al. Current diagnostic and treatment strategies for specific dilated cardiomyopathies: a scientific statement from the american heart association.Circulation. 2016;134(23):e579-e646. doi:10.1161/CIR.0000000000000455
Ache DY, Guamán C, Viñole L, Vignolo G. Infarto agudo de miocardio sin lesiones coronarias obstructivas - MINOCA: un enigma para el cardiólogo clínico. Rev Urug Cardiol. 2020;3(1):77–86.
Mirijello A, Tarli C, Angelo G, Sestito L, Antonelli M, Ferrulli A, et al. European Journal of Internal Medicine Alcoholic cardiomyopathy : What is known and what is not known. Eur J Intern Med [Internet]. 2017;43:1-5. doi:10.1016/j.ejim.2017.06.014
Cihakova D, Rose NR. Chapter 4 Pathogenesis of myocarditis and dilated cardiomyopathy [Internet]. Advances in Inmunology. 2008; 99:95-114. doi:10.1016/S0065-2776(08)00604-4
Katarzyna Q, Gil E, Pawlak A, Gil RJ, Frontczak-baniewicz M, Bil J. The role of invasive diagnostics and its impact on the treatment of dilated cardiomyopathy : a systematic review. Adv Med Sci [Internet]. 2016;61(2):331-343. doi:10.1016/j.advms.2016.07.001
Acuña JMG, Lago AML, González JR. Miocardiopatía dilatada [Internet]. Medicine.
;11(42):2500–6. doi:10.1016/S0304-5412(13)70654-1
Nakatani BT, Minicucci MF, Okoshi K, Okoshi MP. Tachycardia-induced cardiomyopathy. BMJ Case Rep. 2012;2012:bcr2012006587. doi: 10.1136/bcr-2012-006587.
Bolaños Gómez CE, Sanabria Garita A, Garzona Navas A. Miocardiopatía dilatada inducida por hipotiroidismo. Rev costarric cardiol. 2019;21(2):15–20.
Galve Basilio E, Alfonso Manterola F, Ballester Rodés M, Castro Beiras A, Fernández de Soria Pantoja R, Penas Lado M, et al. Guías de práctica clínica de la Sociedad Española de Cardiología en miocardiopatías y miocarditis. Rev Española Cardiol. 2000;53(3):360–93.
Benton RE, Coughlin SS, Tefft C. Predictors of coronary angiography in patients with idiopathic dilated cardiomyopathy : the Washington, DC dilated cardiomyopathy study. J Clin Epidemiol. 1994;47(5):501-11. doi: 10.1016/0895-4356(94)90297-6.
Di Nunzio HJ. Miocardiopatías de etiología no isquémica. En: Rodríguez Granillo GA, Gómez EB, Cademartiri F. TC y RM cardiovascular: fundamentos Clínicos. Buenos Aires: Journal, 2014. pág. 203-249.
Chandra S, Saraf S, Chaudhary G, Dwivedi SK, Narain VS, Sethi R, et al. Prevalence and trends of occult coronary artery disease in patients with dilated cardiomyopathy. Am J Cardiovasc Dis [Internet]. 2020;10(5):557–63. Disponible en: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7811921/
N ́Guetta R, Yao H, Ehouman E, Ekou A, Anzouan-Kacou JB, Coulibaly I, et al. Hallazgos coronarios angiográficos en dilatados miocardiopatía en una población de África subsahariana. Cardiovasc J Afr. 2021;30(3):2021. doi: 10.5830/CVJA-2019-006